2017年8月24日星期四
What are the hazards of vitreous opacity?
(1) vitreous hemorrhage. What are the hazards of vitreous opacity? Vitreous itself without blood vessels, where the bleeding is foreign, especially to see the retina of large blood vessels, high blood pressure, diabetes, retinal vein plugs and other common in the elderly, trauma is also a common cause. Bleeding, such as long-term non-absorption, can occur in the machine, proliferation, leading to retinal detachment.
(2) vitreous inflammatory exudation. Common in uveitis, suppurative endophthalmitis, sympathetic ophthalmia, syphilitic retinal choroiditis and so on. Examination can be found in the vitreous floating point-like, flocculent inflammatory cells, if serious, may have vitreous empyema, eventually leading to traction retinal detachment.
(3) vitreous pigmentation, can be seen in primary retinal detachment, endophthalmitis, trauma and so on. What are the hazards of vitreous opacity? Pigment particles may be derived from the retinal pigment layer or intravitreal hemorrhage.
(4) intraocular tumors. Infants and young children the most common intraocular malignant tumor is retinoblastoma, and reticulocyte sarcoma is found in the elderly, will lead to vitreous opacity.
(5) vitreous parasites in the vitreous body can also lead to vitreous opacity to pigs, tapeworm cercariae most common, although the age group can occur, but there are obvious regional distribution tendencies. What are the hazards of vitreous opacity? Once the cysticercosis release toxins, can cause severe vitreous opacity, retinal detachment.
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What are the symptoms of chalps in children?
1, the course of a slow, generally no obvious symptoms, and no pain sometimes only heavy feeling, due to the oppression of the tumor caused by temporary astigmatism, or mass oppression of the eye caused by a sense of foreign body.
2, eyelid skin can reach one to several different sizes of circular mass, as small as rice, mung bean, large to soybeans, cherries, smooth surface, not with the skin adhesion, clear edges, no tenderness.
3, if the flip eyelid in the mass in the conjunctival surface, can be seen purple or gray-red limitations of the uplift, if secondary infection, can evolve into a sty, in the formation of granulation with the inside is difficult to distinguish.
4, small lumps can be completely absorbed by themselves, or wear their own conjunctiva surface, the contents of the sample, the formation of mushroom-like buds proliferation, this granulation tissue can also be through the meibomian gland discharge pipeline, Shaped proliferation.
5, when there is secondary sense of the occurrence, you can form the internal sty.
6, elderly patients, especially postoperative recurrence, should be removed specimens sent to pathological examination to rule out the possibility of meibomian adenocarcinoma.
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What is the cause of eye bleeding?
1, retinal blood vessels will be abnormal, but there are a variety of different causes, which will lead to the same pathological damage, such as retinal hemorrhage, exudation, microvascular tumor, neovascularization.
2, as well as systemic vascular disease and blood diseases, such as high blood pressure, diabetes and other retinopathy, which will occur bleeding eyes, so as to bring adverse health.
3, there are inflammatory diseases in the life or immune complex violations of the blood vessel wall, we have common diseases with retinal vein inflammation, optic disc vasculitis, etc., we must pay more attention in life.
4, there is a mechanical obstruction, such as thrombosis, etc., which is one of the causes of bleeding eye.
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2017年8月20日星期日
Ametropia is not six of the therapeutic side
1. Chicken liver corn porridge: the first chicken two washed chopped, waxy corn (millet) 60 grams. A total of porridge, add seasoning. For breakfast or snacks, for poor vision or vision loss are often useful.
2. Sheep liver carrot soup: 50 grams of sheep liver slices, carrots, 100 grams are also cut, with the soup plus seasoning, dishes can be served. Eat for the eyesight useful.
3. Wolfberry leaves egg soup: wolfberry leaves 150 grams of clean the chopped, add water to boil, fast cooked into the broken eggs two, cook egg coagulation Serve, add seasoning soup, table, can eat.
4. Cold lily: purslane, lily dish 30 grams (dry 20 grams), 100 grams of purslane also washed. Into the boiling water in the boiling water picked up, with the seasoning salad food, can be served. Anger on the red eye when the red eye paste can often eat.
5. Eyesight dish: Valley Grain 6 grams, grass Cassia 6 grams, Scrophulariaceae 6 grams, 6 grams of green tea, the number of taste or fried drink, a day. Qinggan eyesight, prevention and treatment eyes faint.
6. Thousands of miles of light tea: thousands of miles light 15 to 20 grams, with boiling water simmer for 20 minutes, on behalf of the tea frequency drink. There is heat, detoxification, insecticide, eyesight role.
What are the symptoms of dacryocystitis?
(A) acute dacryocystitis Symptoms: often occurs on the basis of chronic dacryocystitis but also tears without history and sudden occurrence. Clinical manifestations of the lacrimal sac skin swelling and tenderness, and to the lower eyelid, nasal and buccal spread; local pain and to the forehead and maxillary radiation; conjunctival hyperemia edema, tears, conjunctival sac mucus secretions Submandibular lymph nodes. Body chills, fever and discomfort. Inflammation can develop around the formation of peripheral lacrimal duct inflammation, and gradually form an abscess. Pus can be discharged from the small tears, or inward into the ethmoid sinus, but mostly by the skin surface ulceration and the formation of fistula. Fistula healing, but often become the next episode of acute attack.
(B) neonatal dacryocystitis symptoms: neonatal nasolacrimal duct at the lower opening usually before the secretion of tears (about 3 weeks after birth) has been through. Such as blocked by the epithelial debris and did not form a pipeline, or because of bone stenosis, nasal deformity, and affect the process of self-piercing, resulting in atresia, and then produce neonatal dacryocystitis.
(C) chronic dacryocystitis symptoms: is due to nasolacrimal duct obstruction, resulting in tears and lacrimal sac secretions retention of a chronic disease, is also more common external eye disease. It is often the source of suppurative corneal ulcers, trauma and postoperative intraocular infection. With normal drainage function of the lacrimal mucosa epithelium of infectious bacteria have a better resistance, but if the nasolacrimal duct obstruction, causing the accumulation of tears and lacrimal sac secretions, making it the habit of bacteria, resulting in dacryocystitis Of chronic inflammation. The incidence of women is higher than that of men. Nasal diseases (such as rhinitis, sinusitis, nasal septum bending, nasal hypertrophy, etc.) is also an important cause of nasolacrimal duct obstruction. A small number of chronic dacryocystitis cases can spread from conjunctivitis. In addition to tears and secretions increased, the general no other symptoms. Oppression of the lacrimal sac, there are mucous secretions from the tears back to the conjunctival sac, which is based on diagnosis. Reflux of the secretions can also be with pus, which contains a large number of bacteria, such as Staphylococcus aureus, Streptococcus pneumoniae, Streptococcus and so on.
What are the reasons for the eyeballs?
(1) monocular eyeballs, refers to one eye prominent. Why is the eye prominent? Common reason is the eye behind the eyes of the tumor, hematoma, parasites, tuberculosis and so on.
(2) systemic disease caused by limiting the ball prominent, more common in prominent eyes, such as hyperthyroidism, anterior pituitary hyperthyroidism, myasthenia gravis and so on.
(3) inflammation caused by eyeballs, such as orbital cellulitis, whole eye inflammation, eye fascia and so on.
(4) the eye itself caused by the expansion of the prominent, can be seen in the high myopia and intraorbital tumors within the long.
2017年8月16日星期三
What are the treatments for iritis?
Treatment of iritis 1. Drug treatment
There are oral medication, eye drops or subconjunctival injection and other means. Oral medication to give enough to start, in order to quickly control the inflammation, and finally with the minimum amount to maintain the inflammatory activity completely subsided mainly.
Preimplantation of uveitis local drops with 0.5% cortisone or 0.05% dexamethasone, 4 to 5 times a day, or once per hour, recovery period decreased. Sometimes can be under the conjunctival injection can be.
For patients with uveitis or choroiditis, 0.025% dexamethasone can be injected under the conjunctiva or under the fascia of the eye, or combined with systemic administration, with severe hydrocortisone or dexamethasone intravenous infusion once a day, This can be enough to reach the eye tissue.
(1) non-steroidal anti-inflammatory agent sodium salicylate and indomethacin analgesic and anti-inflammatory effect. Main inhibition of uveitis in the anterior chamber of prostaglandin increased in order to achieve anti-inflammatory or antihypertensive effect, commonly used as aspirin.
(2) antibiotics such as suppurative anterior uveitis can be applied locally or systemic broad-spectrum antibiotics.
(3) immunotherapy for severe uveitis and sympathetic ophthalmia, the use of hormones can be considered ineffective when the use of immunosuppressive agents or immune enhancer. To adjust the abnormal immune function, commonly used immunosuppressive agents are: ① cyclophosphamide can be used alone or with steroid therapy. ② B-dimorphine three times a day, even for 2 to 3 weeks, stop iridocyclitis 1 week, and then 1 to 2 courses. ③ pain can be Ning.
Commonly used immunostimulants are levamisole, for immunocompromised patients.
Iriditis treatment 2. Heat or short wave therapy
Expansion of blood vessels, promote blood circulation, strengthen the absorption of inflammation.
3. symptomatic treatment
(1) for secondary glaucoma can be oral administration of acetaminamide to intraocular pressure decreased.
(2) iris penis available iris puncture or iris resection.
(3) on the iris peripheral adhesions caused by secondary glaucoma feasible iris peripheral resection.
(4) on the cataracts can be under the control of inflammation in the cataract surgery.
There are oral medication, eye drops or subconjunctival injection and other means. Oral medication to give enough to start, in order to quickly control the inflammation, and finally with the minimum amount to maintain the inflammatory activity completely subsided mainly.
Preimplantation of uveitis local drops with 0.5% cortisone or 0.05% dexamethasone, 4 to 5 times a day, or once per hour, recovery period decreased. Sometimes can be under the conjunctival injection can be.
For patients with uveitis or choroiditis, 0.025% dexamethasone can be injected under the conjunctiva or under the fascia of the eye, or combined with systemic administration, with severe hydrocortisone or dexamethasone intravenous infusion once a day, This can be enough to reach the eye tissue.
(1) non-steroidal anti-inflammatory agent sodium salicylate and indomethacin analgesic and anti-inflammatory effect. Main inhibition of uveitis in the anterior chamber of prostaglandin increased in order to achieve anti-inflammatory or antihypertensive effect, commonly used as aspirin.
(2) antibiotics such as suppurative anterior uveitis can be applied locally or systemic broad-spectrum antibiotics.
(3) immunotherapy for severe uveitis and sympathetic ophthalmia, the use of hormones can be considered ineffective when the use of immunosuppressive agents or immune enhancer. To adjust the abnormal immune function, commonly used immunosuppressive agents are: ① cyclophosphamide can be used alone or with steroid therapy. ② B-dimorphine three times a day, even for 2 to 3 weeks, stop iridocyclitis 1 week, and then 1 to 2 courses. ③ pain can be Ning.
Commonly used immunostimulants are levamisole, for immunocompromised patients.
Iriditis treatment 2. Heat or short wave therapy
Expansion of blood vessels, promote blood circulation, strengthen the absorption of inflammation.
3. symptomatic treatment
(1) for secondary glaucoma can be oral administration of acetaminamide to intraocular pressure decreased.
(2) iris penis available iris puncture or iris resection.
(3) on the iris peripheral adhesions caused by secondary glaucoma feasible iris peripheral resection.
(4) on the cataracts can be under the control of inflammation in the cataract surgery.
What is the cause of retinal pigment degeneration?
What are the causes of retinal pigment degeneration?
1, myopia: retinitis pigment degeneration What are the reasons? Myopia is easy to produce vitreous degeneration and posterior detachment. Retinal degeneration such as lattice degeneration, paving stone-like degeneration is more likely to see in myopia. Myopia peripheral retinal vulnerability, and the retraction of the vitreous retina, it is easy to lead to retinal detachment.
2, retinal degeneration: some retinal pigment degeneration, such as lattice-like degeneration, frost-like degeneration, paving stone degeneration, particularly easy to form a retinal hole. This is because the denaturation reduces the adhesion of the retina and reduces the traction resistance.
3, trauma: trauma is also one of the reasons for retinal pigment degeneration. In the contusion, the moment of the impact of the eye can temporarily deform the eye, although the eye wall can adapt to external force, but the vitreous can not, at this time the bottom of the vitreous body separated from the ball wall, prone to retinal jaw teeth off. Perforation trauma can directly cause retinal detachment, and late vitreous proliferation can lead to traction detachment.
4, the body of zinc and copper involved in the formation of melanin, copper or rhodopsin regeneration of the necessary material. So that zinc and other trace elements and enzymes metabolic abnormalities may also be retinal pigment degeneration reasons.
1, myopia: retinitis pigment degeneration What are the reasons? Myopia is easy to produce vitreous degeneration and posterior detachment. Retinal degeneration such as lattice degeneration, paving stone-like degeneration is more likely to see in myopia. Myopia peripheral retinal vulnerability, and the retraction of the vitreous retina, it is easy to lead to retinal detachment.
2, retinal degeneration: some retinal pigment degeneration, such as lattice-like degeneration, frost-like degeneration, paving stone degeneration, particularly easy to form a retinal hole. This is because the denaturation reduces the adhesion of the retina and reduces the traction resistance.
3, trauma: trauma is also one of the reasons for retinal pigment degeneration. In the contusion, the moment of the impact of the eye can temporarily deform the eye, although the eye wall can adapt to external force, but the vitreous can not, at this time the bottom of the vitreous body separated from the ball wall, prone to retinal jaw teeth off. Perforation trauma can directly cause retinal detachment, and late vitreous proliferation can lead to traction detachment.
4, the body of zinc and copper involved in the formation of melanin, copper or rhodopsin regeneration of the necessary material. So that zinc and other trace elements and enzymes metabolic abnormalities may also be retinal pigment degeneration reasons.
What are the symptoms of optic nerve atrophy
What are the manifestations of myopic atrophy:
1, optic nerve nipple tilt and myopia: the height of myopia as the back of the eyeball bulge, the optic nerve oblique into the ball. As the side of the nipple (mostly temporal) backward shift, so that the optic disc under the ophthalmoscope lost normal round and oval. And its backward position connected, there are crescent-shaped spots, known as myopia arc, myopia arc contralateral papillary margin slightly brown, realm fuzzy, ridge-like uplift pull the arc. Stretch myopia medial white, sclera exposed, the outer side of the light brown, disappearance of the pigment epithelium, choroidal collapse caused by myofascicular arc lateral state is clear, but often with the posterior pole optic nerve atrophy into a piece. Myopia is located in the temporal lobe of the majority of cases, also located in the temporal or inferior temporal, rare in the nasal or lower side. When the temporal myopia arc outward, upward, downward continue to extend, can be around the entire optic papilla around, as the optic nerve around the choroidal atrophy.
2, leopard striatal fundus (eigroid fundus), the whole eye was slightly dark gray, with choroid plexus due to diffuse optic atrophy, capillary layer and vascular layer decreased or disappeared, orange-red blood vessels exposed, so that the eyes were leopard skin The
3, vision loss, can not be satisfied with the correction. The cause of vision loss is the extensive optic nerve atrophy of the retinal nerve epithelium. Macular hemorrhage can make a sudden loss of vision. Foerster-fuchs spot and macular atrophy after plaque formation, causing severe visual impairment, vitreous opacity to produce floaters. Vitreous is not completely separated from the retina due to incomplete retraction of the traction, and produce flash illusion.
4, the posterior pole of the optic nerve atrophy spots and lacquer-like damage. Choroidal no visible retinal atrophy spots white or yellowish white. Round or map-like, size, the number of different, isolated or integrated into large. Large atrophic spots can be seen in the optic nerve around the atrophic connection, including the optic disc and macula, including a huge atrophy. At the same time there are pigmentation, residual choroidal large blood vessels. Can also be seen in the branch or mesh white or yellow and white lines, lines and blood vessels similar to the lines, different width, the edge is not neat or jagged, similar to the old lacquer crack, so called paint damage.
5, macular part of the beam phase with macular hole. Ophthalmoscopy, the macula can be seen a clear boundary of circular erythema, diameter of about 1 / 3-1 / 2PD. Erythema around the contact retina slightly gray, if accompanied by limitations from the time, the surrounding iris. Slit lamp under the microscope, if the beam-like degeneration, the light section has a thin front beam tangent, if the hole, then the line is interrupted. Cracks on the outer wall of the hole and the surrounding retina.
1, optic nerve nipple tilt and myopia: the height of myopia as the back of the eyeball bulge, the optic nerve oblique into the ball. As the side of the nipple (mostly temporal) backward shift, so that the optic disc under the ophthalmoscope lost normal round and oval. And its backward position connected, there are crescent-shaped spots, known as myopia arc, myopia arc contralateral papillary margin slightly brown, realm fuzzy, ridge-like uplift pull the arc. Stretch myopia medial white, sclera exposed, the outer side of the light brown, disappearance of the pigment epithelium, choroidal collapse caused by myofascicular arc lateral state is clear, but often with the posterior pole optic nerve atrophy into a piece. Myopia is located in the temporal lobe of the majority of cases, also located in the temporal or inferior temporal, rare in the nasal or lower side. When the temporal myopia arc outward, upward, downward continue to extend, can be around the entire optic papilla around, as the optic nerve around the choroidal atrophy.
2, leopard striatal fundus (eigroid fundus), the whole eye was slightly dark gray, with choroid plexus due to diffuse optic atrophy, capillary layer and vascular layer decreased or disappeared, orange-red blood vessels exposed, so that the eyes were leopard skin The
3, vision loss, can not be satisfied with the correction. The cause of vision loss is the extensive optic nerve atrophy of the retinal nerve epithelium. Macular hemorrhage can make a sudden loss of vision. Foerster-fuchs spot and macular atrophy after plaque formation, causing severe visual impairment, vitreous opacity to produce floaters. Vitreous is not completely separated from the retina due to incomplete retraction of the traction, and produce flash illusion.
4, the posterior pole of the optic nerve atrophy spots and lacquer-like damage. Choroidal no visible retinal atrophy spots white or yellowish white. Round or map-like, size, the number of different, isolated or integrated into large. Large atrophic spots can be seen in the optic nerve around the atrophic connection, including the optic disc and macula, including a huge atrophy. At the same time there are pigmentation, residual choroidal large blood vessels. Can also be seen in the branch or mesh white or yellow and white lines, lines and blood vessels similar to the lines, different width, the edge is not neat or jagged, similar to the old lacquer crack, so called paint damage.
5, macular part of the beam phase with macular hole. Ophthalmoscopy, the macula can be seen a clear boundary of circular erythema, diameter of about 1 / 3-1 / 2PD. Erythema around the contact retina slightly gray, if accompanied by limitations from the time, the surrounding iris. Slit lamp under the microscope, if the beam-like degeneration, the light section has a thin front beam tangent, if the hole, then the line is interrupted. Cracks on the outer wall of the hole and the surrounding retina.
2017年8月5日星期六
What are the hazards of ptosis?
(1) trauma damage to the oculomotor nerve or levator palpebral muscle, Müller muscle, can cause traumatic ptosis.
(2) the disease of the eyelid itself, such as severe trachoma, eyelid tumors, making the weight of the eyelids caused by mechanical ptosis.
(3) no eyeballs, small eyeballs, atrophy of the eye and a variety of reasons leading to decreased orbital fat or orbital content, can cause false ptosis.
(2) the disease of the eyelid itself, such as severe trachoma, eyelid tumors, making the weight of the eyelids caused by mechanical ptosis.
(3) no eyeballs, small eyeballs, atrophy of the eye and a variety of reasons leading to decreased orbital fat or orbital content, can cause false ptosis.
What is the principle of eating eye disease?
Glomerulosclerosis, common retinal pigment epithelium, retinal vein inflammation, choroiditis, etc .; heterozygous retinopathy more common ischemic optic neuropathy, central serous chorioretinopathy Lesions, retinal vascular occlusion, optic atrophy, age-related macular degeneration.
Meat is mainly used for the recovery of ocular fundus, or thermal retinopathy of the recovery period of adjuvant therapy. Beef and lambs have the effect of nourishing and nourishing eyesight, liver, chicken, duck kidney essence and blood, pork, eggs and duck eggs liver and kidney Yin Yin, fish are kidney and Qi, Ziyin raise the role. But this kind of food should eat less meals, not overeating.
Plants are used for adjuvant therapy of hot eye disease, such as melon, loofah, bitter gourd, amaranth, celery, mung bean, red bean, kelp, etc. with detoxification, water swelling, blood circulation, and yellow rice, sorghum rice , Millet, soybeans, black sesame seeds, fungus can be Qi and blood eyesight. Although these foods are mainly used for adjuvant treatment of thermal ocular fundus disease, but the lack of ocular fundus deficiency can also be eaten.
In addition to providing common vitamins, some fruits are also very suitable for hot retinopathy, such as pears, apples, oranges, pineapples, peaches, apricots, watermelons and so on. For the ocular fundus disease, it is best to eat chestnuts, walnuts, lotus heart, longan, red dates and so on.
Meat is mainly used for the recovery of ocular fundus, or thermal retinopathy of the recovery period of adjuvant therapy. Beef and lambs have the effect of nourishing and nourishing eyesight, liver, chicken, duck kidney essence and blood, pork, eggs and duck eggs liver and kidney Yin Yin, fish are kidney and Qi, Ziyin raise the role. But this kind of food should eat less meals, not overeating.
Plants are used for adjuvant therapy of hot eye disease, such as melon, loofah, bitter gourd, amaranth, celery, mung bean, red bean, kelp, etc. with detoxification, water swelling, blood circulation, and yellow rice, sorghum rice , Millet, soybeans, black sesame seeds, fungus can be Qi and blood eyesight. Although these foods are mainly used for adjuvant treatment of thermal ocular fundus disease, but the lack of ocular fundus deficiency can also be eaten.
In addition to providing common vitamins, some fruits are also very suitable for hot retinopathy, such as pears, apples, oranges, pineapples, peaches, apricots, watermelons and so on. For the ocular fundus disease, it is best to eat chestnuts, walnuts, lotus heart, longan, red dates and so on.
What are the complications of trichiasis?
⑴ conjunctival inflammation: due to repeated stimulation of the eyelashes conjunctiva and cornea, so conjunctivitis is more difficult to cure, often easy to repeated attacks, tears, secretions often appear. If the conjunctival inflammation repeated and long-term unhealed, and some patients even appear conjunctival scar, a few will appear local symblepharon.
⑵ corneal changes: trichiasis can cause corneal epithelial punctate or diffuse injury, corneal epithelial shedding, corneal opacities, corneal pannus, corneal epithelial keratosis, corneal ulcers, corneal leukoplakia, and thus affect vision.
⑶ other: In addition to corneal lesions affect vision, the inverted eyelashes sometimes cause astigmatism can also affect the vision, in children if the astigmatism is obvious, then a small part can also cause amblyopia.
⑵ corneal changes: trichiasis can cause corneal epithelial punctate or diffuse injury, corneal epithelial shedding, corneal opacities, corneal pannus, corneal epithelial keratosis, corneal ulcers, corneal leukoplakia, and thus affect vision.
⑶ other: In addition to corneal lesions affect vision, the inverted eyelashes sometimes cause astigmatism can also affect the vision, in children if the astigmatism is obvious, then a small part can also cause amblyopia.
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